Mechanical rejuvenation of senescent stem cells and aged bone via chromatin remodeling

Bone aging compromises skeletal integrity and increases vulnerability to osteoporosis and other age-related disorders, underscoring the need for new therapeutic strategies. Although pharmacological and genetic approaches have been widely explored, how cellular mechanical remodeling contributes to bone aging remains unclear. Here, we find that senescent bone marrow stem cells show markedly reduced intracellular force and impaired mechanical behavior. Moderate mechanical stimulation in cell culture and in mice restores cellular force, increases chromatin accessibility at the FOXO1 locus, activates its expression, and reverses cellular senescence and bone aging. These mechanical interventions also improve physical performance in aged female mice and show a tendency to reduce systemic inflammation, whereas excessive force induces chromatin overextension and DNA damage, indicating the necessity of precise force control. In this work, we show that optimized mechanical stimulation provides a simple and effective strategy to counteract age-related bone deterioration and systemic inflammation, offering potential for clinical translation.

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Publication Details

Journal
Nature Communications
Published
2026-01-13
DOI
https://doi.org/10.1038/s41467-026-68387-3
Citations
2
Primary Topic
Telomeres, Telomerase, and Senescence
Type
article
Field-Weighted Citation Impact
65.68

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article

Mechanical rejuvenation of senescent stem cells and aged bone via chromatin remodeling

Xiaojing Liu, Qiang Wei, Zhonghan Li, Li Liao et al.
2 citations
Nature Communications
Telomeres, Telomerase, and Senescence
65.68
article

Mechanical rejuvenation of senescent stem cells and aged bone via chromatin remodeling

Xiaojing Liu, Qiang Wei, Zhonghan Li, Li Liao, Yuanxin Ye
article en
2 citations

Abstract

Bone aging compromises skeletal integrity and increases vulnerability to osteoporosis and other age-related disorders, underscoring the need for new therapeutic strategies. Although pharmacological and genetic approaches have been widely explored, how cellular mechanical remodeling contributes to bone aging remains unclear. Here, we find that senescent bone marrow stem cells show markedly reduced intracellular force and impaired mechanical behavior. Moderate mechanical stimulation in cell culture and in mice restores cellular force, increases chromatin accessibility at the FOXO1 locus, activates its expression, and reverses cellular senescence and bone aging. These mechanical interventions also improve physical performance in aged female mice and show a tendency to reduce systemic inflammation, whereas excessive force induces chromatin overextension and DNA damage, indicating the necessity of precise force control. In this work, we show that optimized mechanical stimulation provides a simple and effective strategy to counteract age-related bone deterioration and systemic inflammation, offering potential for clinical translation.

Nature CommunicationsVol. 17(1)
Ministry of Education of the People's Republic of China (CN), Sichuan University (CN), West China Hospital of Sichuan University (CN), Ingenierie des Materiaux polymeres (FR)
National Natural Science Foundation of China
Good health and well-being
Openalex Percentile: Top 0%
Telomeres, Telomerase, and Senescence
65.68
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Mechanical rejuvenation of senescent stem cells and aged bone via chromatin remodeling — Xiaojing Liu, Qiang Wei, et al. · Nature Communications (2026) | TGRS Research Map | TGRS