Targeting Triglycerides in Cardiovascular Disease Prevention: Evidence, Mechanisms, and Emerging Therapies

Emerging data from Mendelian randomization studies and large clinical cohorts support a causal link between elevated remnant lipoproteins and atherosclerotic CVD, in which apolipoprotein B may be the principal driver. Although traditional triglyceride-lowering agents have produced mixed results on cardiovascular outcomes, emerging therapies-such as ApoC-III and ANGPTL3 inhibitors-show robust lipid-lowering effects, while selective PPAR modulators have thus far not demonstrated cardiovascular benefit. However, outcome data remain limited. Residual CVD risk persists despite aggressive LDL-C reduction, especially in patients with diabetes, metabolic syndrome, or chronic kidney disease. Selective TG-lowering strategies targeting TRLs-especially those that decrease apolipoprotein B-may provide clinical benefit in high-risk phenotypes. Ongoing trials will clarify whether these promising agents confer meaningful cardiovascular protection and warrant integration into future guidelines.

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Publication Details

Journal
Current Cardiology Reports
Published
2026-01-03
DOI
https://doi.org/10.1007/s11886-025-02337-1
Primary Topic
Lipid metabolism and disorders
Type
article
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article

Targeting Triglycerides in Cardiovascular Disease Prevention: Evidence, Mechanisms, and Emerging Therapies

Salim S. Virani, Annalisa Filtz, Leandro N. Slipczuk, Michael D. Shapiro et al.
Current Cardiology Reports
Lipid metabolism and disorders
article

Targeting Triglycerides in Cardiovascular Disease Prevention: Evidence, Mechanisms, and Emerging Therapies

Salim S. Virani, Annalisa Filtz, Leandro N. Slipczuk, Michael D. Shapiro, P. Toth, Usman Alam, Toshiki Kuno, Badimón Jj, Sheetal Vasundara Mathai, Carl J. Lavie, Deepak L. Bhatt, Allan D. Sniderman
article en

Abstract

Emerging data from Mendelian randomization studies and large clinical cohorts support a causal link between elevated remnant lipoproteins and atherosclerotic CVD, in which apolipoprotein B may be the principal driver. Although traditional triglyceride-lowering agents have produced mixed results on cardiovascular outcomes, emerging therapies-such as ApoC-III and ANGPTL3 inhibitors-show robust lipid-lowering effects, while selective PPAR modulators have thus far not demonstrated cardiovascular benefit. However, outcome data remain limited. Residual CVD risk persists despite aggressive LDL-C reduction, especially in patients with diabetes, metabolic syndrome, or chronic kidney disease. Selective TG-lowering strategies targeting TRLs-especially those that decrease apolipoprotein B-may provide clinical benefit in high-risk phenotypes. Ongoing trials will clarify whether these promising agents confer meaningful cardiovascular protection and warrant integration into future guidelines.

Current Cardiology ReportsVol. 28(1)
Aga Khan University (PK), Albert Einstein College of Medicine (US), Beth Israel Deaconess Medical Center (US), Montefiore Medical Center (US), Harvard University (US), Johns Hopkins University (US), Johns Hopkins Medicine (US), McGill University Health Centre (CA), Royal Victoria Hospital (CA), Mount Sinai Hospital (US), Aga Khan University Nairobi (KE), Montefiore Health System (US), CGH Medical Center (US), Wake Forest University (US), Icahn School of Medicine at Mount Sinai (US)
Good health and well-being
Openalex Percentile: Top 98%
Lipid metabolism and disorders
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