Trajectories of CSF and plasma MTBR‐tau 243 and phosphorylated‐tau species across the Alzheimer's disease continuum
CSF or plasma p-tau217, p-tau205, and MTBR-tau243 provide information about different biological events in the disease cascade (Figure 3), which can benefit clinical trials and patient management in clinical practice.
Authors
- Tobey J. Betthauser (ORCID: https://orcid.org/0000-0001-8856-1352)
- Rik Ossenkoppele (ORCID: https://orcid.org/0000-0003-1584-7477)
- Olof T. Strandberg
- Gemma Salvadó (ORCID: https://orcid.org/0000-0002-5210-9230)
- Kanta Horie (ORCID: https://orcid.org/0000-0001-7736-2614)
- Oskar H. Hansson
- Lyduine E. Collij
- Niklas Mattsson‐Carlgren
- Sebastian Palmqvist
- Randall J. Bateman
- Ruben Smith
- Shorena Janelidze
- Nicolas R. Barthélemy
- Suzanne E. Schindler
Institutions
- University of Wisconsin–Madison (US)
- Lund University (SE)
- Washington University in St. Louis (US)
- Pasqual Maragall Foundation (ES)
- Amsterdam Neuroscience (NL)
- Hope Center for Neurological Disorders (US)
- Barcelonaβeta Brain Research Center (ES)
- Skåne University Hospital (SE)
- Wisconsin Division of Public Health (US)
- Eisai (United States) (US)
- Amsterdam University Medical Centers (NL)
- Vrije Universiteit Amsterdam (NL)
Publication Details
- Journal
- Alzheimer s & Dementia
- Published
- 2025-12-01
- DOI
- https://doi.org/10.1002/alz70856_098557
- Primary Topic
- Dementia and Cognitive Impairment Research
- Type
- article
- Field-Weighted Citation Impact
- 0.00