Nafamostat Mesylate Protects Against Acute Renal Ischemia–Reperfusion Injury by Alleviating Mitochondrial Dysfunction, Inhibiting Ferroptosis, and Regulating Proximal Tubular Cells: A Multi‐Omics Analysis

NM protects against AKI by reducing reactive oxygen species, upregulating fatty acids, and regulating ferroptosis signaling pathways in proximal tubular cells.

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Publication Details

Journal
Therapeutic Apheresis and Dialysis
Published
2025-12-23
DOI
https://doi.org/10.1002/1744-9987.70112
Primary Topic
Ferroptosis and cancer prognosis
Type
article
Field-Weighted Citation Impact
0.00
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article

Nafamostat Mesylate Protects Against Acute Renal Ischemia–Reperfusion Injury by Alleviating Mitochondrial Dysfunction, Inhibiting Ferroptosis, and Regulating Proximal Tubular Cells: A Multi‐Omics Analysis

Santao Ou, Hanfeng Li, YuHan Wang, Y Li et al.
Therapeutic Apheresis and Dialysis
Ferroptosis and cancer prognosis
article

Nafamostat Mesylate Protects Against Acute Renal Ischemia–Reperfusion Injury by Alleviating Mitochondrial Dysfunction, Inhibiting Ferroptosis, and Regulating Proximal Tubular Cells: A Multi‐Omics Analysis

Santao Ou, Hanfeng Li, YuHan Wang, Y Li, B. Chen, Huameng Xing, jiang liu, Yuxuan Wu, Qiaoyu Wang, Weihua Wu, Lan Huang, Ling Xue, Dongmei Zhang
article en

Abstract

NM protects against AKI by reducing reactive oxygen species, upregulating fatty acids, and regulating ferroptosis signaling pathways in proximal tubular cells.

Therapeutic Apheresis and DialysisVol. 30(2)
Chengdu Medical College (CN), Southwest Medical University (CN), Affiliated Hospital of Southwest Medical University (CN), Chengdu Fifth People's Hospital (CN), Zigong First People's Hospital (CN)
Openalex Percentile: Top 28%
Ferroptosis and cancer prognosis
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