Spontaneous cardiac rupture as the initial presentation of acute myeloid leukaemia complicated by malignant lactic acidosis: a case report

This case underscores three key clinical considerations:(1) In instances of LVFWR without culprit coronary artery occlusion, the possibility of leukaemia-associated myocardial pathology should be actively evaluated.(2) For oozing-type LVFWR lacking a clearly identifiable rupture site, sutureless repair may represent a feasible and effective surgical option.(3) The onset or progression of lactic acidosis in the setting of marked leucocytosis constitutes a medical emergency, warranting prompt initiation of cytoreductive therapy and empiric thiamine supplementation.