FAK-Activated Mucosal Healing Promotes Resistance to Reinjury
FAK activation accelerates ischemic ulcer healing, in part by enhancing angiogenesis. Moreover, FAK activation during an initial injury reduces susceptibility to recurrent NSAID-induced intestinal injury, perhaps because it promotes initial higher-quality ulcer repair.
Authors
- Vadim J. Gurvich (ORCID: https://orcid.org/0000-0002-5420-1639)
- Marc D. Basson (ORCID: https://orcid.org/0000-0001-9696-2789)
- Louis Boafo Kwantwi (ORCID: https://orcid.org/0000-0002-4015-2127)
- Sema Oncel (ORCID: https://orcid.org/0000-0001-5551-2271)
- Guiming Liu (ORCID: https://orcid.org/0009-0008-7433-7075)
- Emilie E. Vomhof-DeKrey
- Ricardo Gallardo-Macias
Institutions
- University of Minnesota (US)
- University of North Dakota (US)
- Northeast Ohio Medical University (US)
Publication Details
- Journal
- Cells
- Published
- 2025-12-22
- DOI
- https://doi.org/10.3390/cells15010016
- Primary Topic
- Cell Adhesion Molecules Research
- Type
- article
- Field-Weighted Citation Impact
- 0.00