Novel mechanism of tumor metastasis: DDX11–ATAD5 interaction mediates epithelial–mesenchymal transition to promote gallbladder cancer progression

This study reveals a novel molecular mechanism whereby DDX11-ATAD5 interaction promotes GBC progression through EMT activation, providing potential therapeutic targets for GBC diagnosis and treatment.

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Publication Details

Journal
CytoJournal
Published
2025-12-11
DOI
https://doi.org/10.25259/cytojournal_42_2025
Primary Topic
Ferroptosis and cancer prognosis
Type
article
Field-Weighted Citation Impact
0.00
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article

Novel mechanism of tumor metastasis: DDX11–ATAD5 interaction mediates epithelial–mesenchymal transition to promote gallbladder cancer progression

CytoJournal
Ferroptosis and cancer prognosis
article

Novel mechanism of tumor metastasis: DDX11–ATAD5 interaction mediates epithelial–mesenchymal transition to promote gallbladder cancer progression

article en

Abstract

This study reveals a novel molecular mechanism whereby DDX11-ATAD5 interaction promotes GBC progression through EMT activation, providing potential therapeutic targets for GBC diagnosis and treatment.

CytoJournalVol. 22
Ruijin Hospital (CN), Central Hospital of Putuo District (CN), Jining Medical University (CN)
Openalex Percentile: Top 30%
Ferroptosis and cancer prognosis
DOIOpen Access PDFOpenAlex
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Novel mechanism of tumor metastasis: DDX11–ATAD5 interaction mediates epithelial–mesenchymal transition to promote gallbladder cancer progression · CytoJournal (2025) | TGRS Research Map | TGRS