The Wnt1G177C mutation impairs mechanically induced bone formation via Nr4a2

Objectives: Wnt1 plays a crucial role in mediating the skeletal response to mechanical load, but its mechanisms, especially concerning the Wnt1 mutation associated with osteogenesis imperfecta type XV (OI-XV), remain elusive. We investigated the impact of the Wnt1G177C loss-of-function mutation during [for full text, please go to the a.m. URL]

Institutions

Publication Details

Journal
German Medical Science (German Research Foundation)
Published
2024-10-21
DOI
https://doi.org/10.3205/24dkou045
Primary Topic
Connective tissue disorders research
Type
article
Field-Weighted Citation Impact
0.00
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article

The Wnt1G177C mutation impairs mechanically induced bone formation via Nr4a2

German Medical Science (German Research Foundation)
Connective tissue disorders research
article

The Wnt1G177C mutation impairs mechanically induced bone formation via Nr4a2

article en

Abstract

Objectives: Wnt1 plays a crucial role in mediating the skeletal response to mechanical load, but its mechanisms, especially concerning the Wnt1 mutation associated with osteogenesis imperfecta type XV (OI-XV), remain elusive. We investigated the impact of the Wnt1G177C loss-of-function mutation during [for full text, please go to the a.m. URL]

German Medical Science (German Research Foundation)
Universität Hamburg (DE), University Medical Center Hamburg-Eppendorf (DE)
Openalex Percentile: Top 54%
Connective tissue disorders research
DOIOpen Access PDFOpenAlex
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The Wnt1G177C mutation impairs mechanically induced bone formation via Nr4a2 · German Medical Science (German Research Foundation) (2024) | TGRS Research Map | TGRS