Retinal pericyte destabilization in topical glucocorticoid exposure: Mechanistic pathways to ocular terrain collapse

Reframes tGCS-induced ocular injury as a terrain collapse syndrome driven by retinal pericyte destabilization, integrating vascular, immune, and redox mechanisms to unify pathology and guide new diagnostic and therapeutic approaches.

Publication Details

Journal
Zenodo (CERN European Organization for Nuclear Research)
Published
2025-08-09
DOI
https://doi.org/10.5281/zenodo.16787067
Primary Topic
Mathematics, Computing, and Information Processing
Type
article
Field-Weighted Citation Impact
0.00
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article

Retinal pericyte destabilization in topical glucocorticoid exposure: Mechanistic pathways to ocular terrain collapse

Zenodo (CERN European Organization for Nuclear Research)
Mathematics, Computing, and Information Processing
article

Retinal pericyte destabilization in topical glucocorticoid exposure: Mechanistic pathways to ocular terrain collapse

article en

Abstract

Reframes tGCS-induced ocular injury as a terrain collapse syndrome driven by retinal pericyte destabilization, integrating vascular, immune, and redox mechanisms to unify pathology and guide new diagnostic and therapeutic approaches.

Zenodo (CERN European Organization for Nuclear Research)
Sustainable cities and communities
Openalex Percentile: Top 53%
Mathematics, Computing, and Information Processing
DOIOpen Access PDFOpenAlex
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Retinal pericyte destabilization in topical glucocorticoid exposure: Mechanistic pathways to ocular terrain collapse · Zenodo (CERN European Organization for Nuclear Research) (2025) | TGRS Research Map | TGRS