Physiological Effects of Occupational Stress on the Cardiovascular System

Occupational stress is documented to have catastrophic effects on cardiovascular health in the workplace. The physiological effect of chronic stress has been shown to alter the HPA axis and the sympathetic nervous system, in proving higher cortisol levels, higher catecholamine levels, and an imbalance of the autonomic nervous system. These physiological changes cause hypertension, endothelial dysfunction, atherosclerosis and reduced HRV which in turn predispose individuals to CVDs. In addition, there are inflammation and oxidative stress that enhance arterial plaque formation as well as thrombogenesis and increase cardiovascular morbidity and mortality. This study examines how occupational stress promotes cardiovascular disease through autonomic and hormonal systems and through biochemical stress responses. Conclusions derived from the study provides substantial support for measures such as stress control and organizational approaches to decrease these risks.