The quantification of EdU-488 assays and transwell assays after the employment of NF-κB inhibitor.

<p>RBE and HuCCT1 cells were pretreated with 10 μM BAY 11–7082 (a NF-κB inhibitor) for 1 h, followed by stimulation with 10 μg/ml of <i>Cs</i>EVs for 24 h. EdU-488 assays <b>(A, B)</b> and Transwell assays <b>(C, D)</b> revealed that NF-κB inhibitor markedly reversed the <i>Cs</i>EVs-induced malignant proliferation and migration in both RBE and HuCCT1 cells.</p> <p>(DOCX)</p>

Publication Details

Journal
Figshare
Published
2024-10-28
DOI
https://doi.org/10.1371/journal.pntd.0012545.s002
Primary Topic
NF-κB Signaling Pathways
Type
article
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0.00
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article

The quantification of EdU-488 assays and transwell assays after the employment of NF-κB inhibitor.

Figshare
NF-κB Signaling Pathways
article

The quantification of EdU-488 assays and transwell assays after the employment of NF-κB inhibitor.

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Abstract

<p>RBE and HuCCT1 cells were pretreated with 10 μM BAY 11–7082 (a NF-κB inhibitor) for 1 h, followed by stimulation with 10 μg/ml of <i>Cs</i>EVs for 24 h. EdU-488 assays <b>(A, B)</b> and Transwell assays <b>(C, D)</b> revealed that NF-κB inhibitor markedly reversed the <i>Cs</i>EVs-induced malignant proliferation and migration in both RBE and HuCCT1 cells.</p> <p>(DOCX)</p>

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The quantification of EdU-488 assays and transwell assays after the employment of NF-κB inhibitor. · Figshare (2024) | TGRS Research Map | TGRS