Auxin treatment in the absence of RBM-26::Scarlet::AID does not cause PLM axon termination defect or beading phenotype.

<p>L4 Wild type or Neuronal TIR-1 (without AID tagged RBM-26) were placed on 4 mM synthetic Auxin-coated plates, allowed to lay eggs and their L3 progenies were analyzed for (A) PLM termination defect and (B) beading phenotype; “<i>n</i>” is 132 for wt and 138 for Neuronal TIR-1. N.S. = Not Significant (Z-test for proportions). Error bars represent the standard error of the proportion. Underlying data can be found in <a href="http://www.plosbiology.org/article/info:doi/10.1371/journal.pbio.3002876#pbio.3002876.s017" target="_blank">S2 Data</a>.</p> <p>(PDF)</p>