Additional file 1 of SNORA37/CMTR1/ELAVL1 feedback loop drives gastric cancer progression via facilitating CD44 alternative splicing

Additional file 1: Figure S1. Expression profiles of snoRNAs in gastric cancer tissues. Figure S2. Oncogenic roles of SNORA37 in gastric cancer. Figure S3. Expression profiles and alternative splicing of CD44 and PRMT2 in gastric cancer. Figure S4. SNORA37 interacts with CMTR1. Figure S5. CMTR1 facilitates nuclear retention of oncogenic ELAVL1 in gastric cancer cells. Figure S6. SNORA37 enhances the interaction of CMTR1 with ELAVL1. Figure S7. SNORA37 facilitates alternative splicing of CD44 via CMTR1 and ELAVL1. Figure S8. SNORA37 facilitates gastric cancer progression via enhancing interaction of CMTR1 with ELAVL1. Table S1. Primer sets used for RT-PCR, RIP, and probe. Table S2. Primer sets used for constructs. Table S3. Oligonucleotide sets for short hairpin RNAs. Table S4. Differentially expressed snoRNAs in RNA-seq assay. Table S5. Alteration of splicing events in MKN-45 cells upon SNORA37 over-expression. Table S6. Alteration of splicing events in gastric tissues. Table S7. Mass spectrometry analysis of proteins pulled down by SNORA37.