Additional file 1 of Single-cell transcriptomics identify a novel macrophage population associated with bone invasion in pituitary neuroendocrine tumors

Supplementary Material 1: Supplementary Figure S1: Basic information of the single-cell RNA-seq data. Supplementary Figure S2: Cluster characterization of myeloid cells. Supplementary Figure S3: CX3CR1+ TREM2+ Mo-TAM is the source of TNF in BI-PitNETs. Supplementary Figure S4: Cell–cell communication between TNFα+ TAMs and CD14+ monocytes. Supplementary Figure S5: Spatial distribution of IL34+ tumor. Supplementary Figure S6: Characteristics of T cells in PitNET. Supplementary Figure S7: Landscape of fibroblasts in PitNET.

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Publication Details

Journal
Figshare
Published
2025-01-01
DOI
https://doi.org/10.6084/m9.figshare.28283331.v1
Primary Topic
Neuroinflammation and Neurodegeneration Mechanisms
Type
article
Field-Weighted Citation Impact
0.00
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article

Additional file 1 of Single-cell transcriptomics identify a novel macrophage population associated with bone invasion in pituitary neuroendocrine tumors

Figshare
Neuroinflammation and Neurodegeneration Mechanisms
article

Additional file 1 of Single-cell transcriptomics identify a novel macrophage population associated with bone invasion in pituitary neuroendocrine tumors

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Abstract

Supplementary Material 1: Supplementary Figure S1: Basic information of the single-cell RNA-seq data. Supplementary Figure S2: Cluster characterization of myeloid cells. Supplementary Figure S3: CX3CR1+ TREM2+ Mo-TAM is the source of TNF in BI-PitNETs. Supplementary Figure S4: Cell–cell communication between TNFα+ TAMs and CD14+ monocytes. Supplementary Figure S5: Spatial distribution of IL34+ tumor. Supplementary Figure S6: Characteristics of T cells in PitNET. Supplementary Figure S7: Landscape of fibroblasts in PitNET.

Figshare
Capital University (US), Capital Medical University (CN), Chinese Academy of Sciences (CN), University of California San Diego (US), Beijing Tian Tan Hospital (CN)
Openalex Percentile: Top 51%
Neuroinflammation and Neurodegeneration Mechanisms
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