<b>Hyperoxia-induced senescence in fetal airway smooth muscle cells: Role of mitochondrial reactive oxygen species and endoplasmic reticulum stress</b>

Supplemental file 1: representative digital blots from the capillary JESS showing the specificity of the used antibodies targeting the selected ER stress, senescence, pro- and anti-apoptosis, and mitochondria dynamic markers.Supplemental file 2: Effect of Salubrinal [12.5 μM], MitoQ [100 nM] or Mdivi-1 [10 μM] on senescence markers (LMNB1, Cav-1, PAI-1) and pro-apoptotic marker (caspase-3), in hyperoxia-exposed fASM.