Diagnosing Invasive Ductal Carcinoma Prediction in Breast Cancer
Breast cancer is one of the most common and life-threatening diseases affecting women worldwide,with invasive ductal carcinoma (IDC) being the most frequently diagnosed type, accounting for over 80%of all breast cancer cases. Early and accurate detection is critical in improving patient survival rates, astimely intervention significantly enhances treatment success. However, traditional diagnostic methods relyheavily on pathologists manually analyzing histology slides, which is both time-consuming and subject tohuman variability, leading to potential delays or misdiagnoses. To address these challenges, this researchexplores the use of machine learning-based classification models to automate and enhance the detection ofIDC in breast histology images.To classify IDC presence, the suggested method involves preprocessinghistology images, identifying important morphological features, and using supervised learning algorithms.The goal is to develop a robust model that can generalize well to real-world clinical applications, reducingthe dependency on manual diagnosis and minimizing human error.In addition to classification accuracy, thisstudy also examines the interpretability and scalability of different machine-learning approaches, ensuringthat the developed model is reliable and practical for integration into existing medical workflows. Byautomating IDC detection, this research aims to enhance diagnostic efficiency, support early intervention,and contribute to the advancement of AI-assisted pathology. The results of this study could enhance breastcancer screening, help physicians make better decisions, and eventually raise the likelihood of earlydetection and successful treatment, which would benefit patients.
Publication Details
- Journal
- Zenodo (CERN European Organization for Nuclear Research)
- Published
- 2025-04-27
- DOI
- https://doi.org/10.5281/zenodo.15290827
- Primary Topic
- AI in cancer detection
- Type
- article
- Field-Weighted Citation Impact
- 0.00