Mechanism of b-catenin in Pulmonary Fibrosis Following SARS-CoV-2 Infection

In this study, we stably expressed human angiotensin-converting enzyme 2 (hACE2) in the human non-small cell lung cancer cell line A549 and investigated the role of b-catenin in SARS-CoV-2 infection. Following knocked down of b-catenin, a canonical component of the Wnt/b-catenin signaling pathway, cells were infected with SARS-CoV-2.

Publication Details

Journal: China National GeneBank DataBase
Published: 2025-01-01
DOI: https://doi.org/10.26036/cnp0006698
Primary Topic: Renin-Angiotensin System Studies
Type: article
Field-Weighted Citation Impact: 0.00

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article

Mechanism of b-catenin in Pulmonary Fibrosis Following SARS-CoV-2 Infection

Jan 1, 2025

China National GeneBank DataBase

Renin-Angiotensin System Studies

article

Mechanism of b-catenin in Pulmonary Fibrosis Following SARS-CoV-2 Infection

January 1, 2025article en

Abstract

China National GeneBank DataBase

Good health and well-being

Openalex Percentile: Top 41%

Renin-Angiotensin System Studies

DOI Open Access PDF OpenAlex

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Mechanism of b-catenin in Pulmonary Fibrosis Following SARS-CoV-2 Infection · China National GeneBank DataBase (2025) | TGRS Research Map | TGRS