Potential molecular mechanisms in different PHN models.

<p>The table presents potential mechanisms of PHN models, including information on model types, potential pathways, biofunctions, and other relevant details. “/” means not mentioned in the article. CCL5, C-C motif chemokine ligand 5; CCR5, C-C chemokine receptor type 5; HSV-1, herpes simplex virus type 1; DRG, dorsal root ganglion; TLR4, toll-like receptor 4; TNF, tumor necrosis factor; PHP, pseudohypoparathyroidism; K+, potassium ion; COX, cyclooxygenase; PGE2, prostaglandin E2; EP3, prostaglandin E2 receptor EP3; NMDA, N-Methyl-D-Aspartate; NR2Bs, N-Methyl-D-Aspartate receptor subunit 2B; NOS, nitric oxide synthase; Prmt6, protein arginine methyltransferase 6; cGAS, cyclic GMP-AMP synthase; STING, stimulator of interferon genes; VPM, ventral posterior medial nucleus; VPL, ventral posterior lateral nucleus; P2X7, purinergic receptor P2X, ligand-gated ion channel 7; BBG, brilliant blue G; ER stress, endoplasmic reticulum stress; KCNA2, potassium voltage-gated channel subfamily A member 2; STAT3, signal transducer and activator of transcription 3; pSTAT3, phosphorylated signal transducer and activator of transcription 3; MRC-5, medical research council cell strain 5; TRPV1, transient receptor potential vanilloid 1; NO, nitric oxide; vHPPE, ventriculoperitoneal hydrocephalus; VZV, varicella zoster virus; SNT, syntrophin; H3, histone H3; PWL, paw withdrawal latency; MWT, mechanical withdrawal threshold; TRX, thioredoxin; NP, nucleoside phosphorylase; DNF, brain-derived neurotrophic factor; ASIC3, acid-sensing ion channel 3; PAQR, progestin and AdipoQ receptor; VGAT, vesicular GABA transporter; ERK, extracellular signal-regulated kinase; IE4, immediate early protein 4.</p> <p>(DOCX)</p>