Compound 4f restores the expression of several genes changed by kainic acid (KA) in neuroinflammation, γ-aminobutyric acid (GABA) receptor, glutamate receptor signaling, and endocannabinoid neuronal synapse pathways.

<p><i>P</i> value for KA-injected mice treated with compound 4f compared to KA-injected mice treated with the vehicle. Ratio (KA + vehicle/KA + compound <b>4f</b>): Number of genes significantly changed by compound <b>4f</b> among those genes changed by KA (FC ≤ -1.2 or ≥ 1.2)/number of genes changed by KA (FC ≤ -1.2 or ≥ 1.2).</p> <p>(PDF)</p>

Publication Details

Journal
Figshare
Published
2024-11-21
DOI
https://doi.org/10.1371/journal.pone.0312090.s006
Primary Topic
Neuroscience and Neuropharmacology Research
Type
article
Field-Weighted Citation Impact
0.00
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article

Compound 4f restores the expression of several genes changed by kainic acid (KA) in neuroinflammation, γ-aminobutyric acid (GABA) receptor, glutamate receptor signaling, and endocannabinoid neuronal synapse pathways.

Figshare
Neuroscience and Neuropharmacology Research
article

Compound 4f restores the expression of several genes changed by kainic acid (KA) in neuroinflammation, γ-aminobutyric acid (GABA) receptor, glutamate receptor signaling, and endocannabinoid neuronal synapse pathways.

article en

Abstract

<p><i>P</i> value for KA-injected mice treated with compound 4f compared to KA-injected mice treated with the vehicle. Ratio (KA + vehicle/KA + compound <b>4f</b>): Number of genes significantly changed by compound <b>4f</b> among those genes changed by KA (FC ≤ -1.2 or ≥ 1.2)/number of genes changed by KA (FC ≤ -1.2 or ≥ 1.2).</p> <p>(PDF)</p>

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Neuroscience and Neuropharmacology Research
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Compound 4f restores the expression of several genes changed by kainic acid (KA) in neuroinflammation, γ-aminobutyric acid (GABA) receptor, glutamate receptor signaling, and endocannabinoid neuronal synapse pathways. · Figshare (2024) | TGRS Research Map | TGRS