Hyperglycemia Increased The Expression of CALCRL G Protein-Coupled Receptor in Monocytes from Diabetic Patients.

Hyperglycemia increased the expression of <i>CALCRL</i> GPCR in the monocytes of diabetic patients. The CALCRL receptor may play a key role in regulating vascular inflammation in hyperglycemic conditions. Targeting CALCRL may control inflammation in blood cells.

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Publication Details

Journal
PubMed
Published
2025-12-14
DOI
https://doi.org/10.22074/cellj.2025.2059066.1843
Primary Topic
Diabetes and associated disorders
Type
article
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0.00
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article

Hyperglycemia Increased The Expression of CALCRL G Protein-Coupled Receptor in Monocytes from Diabetic Patients.

PubMed
Diabetes and associated disorders
article

Hyperglycemia Increased The Expression of CALCRL G Protein-Coupled Receptor in Monocytes from Diabetic Patients.

article en

Abstract

Hyperglycemia increased the expression of <i>CALCRL</i> GPCR in the monocytes of diabetic patients. The CALCRL receptor may play a key role in regulating vascular inflammation in hyperglycemic conditions. Targeting CALCRL may control inflammation in blood cells.

PubMedVol. 27(1)
Tarbiat Modares University (IR), Tehran University of Medical Sciences (IR), Ludwig-Maximilians-Universität München (DE)
Openalex Percentile: Top 57%
Diabetes and associated disorders
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Hyperglycemia Increased The Expression of CALCRL G Protein-Coupled Receptor in Monocytes from Diabetic Patients. · PubMed (2025) | TGRS Research Map | TGRS