Dl-3-n-butylphthalide protects against ischemic stroke by enhancing mitochondrial function via MT-CO1 upregulation

NBP protects against ischemia-induced mitochondrial dysfunction partly through MT-CO1 upregulation, supporting MT-CO1 as a potential therapeutic target for mitochondrial function protection in ischemic stroke.

Institutions

Publication Details

Journal
Journal of Stroke and Cerebrovascular Diseases
Published
2025-12-17
DOI
https://doi.org/10.1016/j.jstrokecerebrovasdis.2025.108529
Citations
1
Primary Topic
Neurological Disease Mechanisms and Treatments
Type
article
Field-Weighted Citation Impact
0.84
Controls
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article

Dl-3-n-butylphthalide protects against ischemic stroke by enhancing mitochondrial function via MT-CO1 upregulation

1 citations
Journal of Stroke and Cerebrovascular Diseases
Neurological Disease Mechanisms and Treatments
0.84
article

Dl-3-n-butylphthalide protects against ischemic stroke by enhancing mitochondrial function via MT-CO1 upregulation

article en
1 citations

Abstract

NBP protects against ischemia-induced mitochondrial dysfunction partly through MT-CO1 upregulation, supporting MT-CO1 as a potential therapeutic target for mitochondrial function protection in ischemic stroke.

Journal of Stroke and Cerebrovascular DiseasesVol. 35(2)
Hunan City University (CN), Yantai Laiyang Central Hospital (CN)
Openalex Percentile: Top 19%
Neurological Disease Mechanisms and Treatments
0.84
DOIOpen Access PDFOpenAlex
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Dl-3-n-butylphthalide protects against ischemic stroke by enhancing mitochondrial function via MT-CO1 upregulation · Journal of Stroke and Cerebrovascular Diseases (2025) | TGRS Research Map | TGRS