The Role of Angiopoietin-2 in Post-Burn Pneumonia
These findings suggest that burn injury increases Angiopoetin-2 and downstream signaling in the lungs, which may contribute to post-burn pulmonary dysfunction. Further studies are necessary to understand if modulating the Ang-TIE2 axis can protect against pneumonia post-burn.
Authors
- Nadja García (ORCID: https://orcid.org/0000-0001-6964-577X)
- Madison M. Tschann (ORCID: https://orcid.org/0009-0000-1622-092X)
- Anna K. Brady
- Kevin E Galicia (ORCID: https://orcid.org/0000-0001-7930-897X)
- John Kubasiak (ORCID: https://orcid.org/0000-0003-1446-3391)
- Irene Helenowski (ORCID: https://orcid.org/0000-0003-1606-9013)
- Mashkoor A. Choudhry (ORCID: https://orcid.org/0000-0002-1152-7145)
- Connor O Guzior
- Mary Grace Murray (ORCID: https://orcid.org/0000-0002-2533-3050)
- Ryan M. Johnson (ORCID: https://orcid.org/0000-0003-1809-6023)
- Richard P. Gonzalez
- Abigail Plum (ORCID: https://orcid.org/0009-0007-4481-411X)
- Madison D Kipp (ORCID: https://orcid.org/0009-0008-8678-725X)
Institutions
- Loyola University Medical Center (US)
- Loyola University Chicago (US)
Publication Details
- Journal
- European Burn Journal
- Published
- 2025-12-19
- DOI
- https://doi.org/10.3390/ebj7010001
- Primary Topic
- Angiogenesis and VEGF in Cancer
- Type
- article
- Field-Weighted Citation Impact
- 0.00