GWAS and Replication Analysis of Apparent Treatment-Resistant Hypertension
We identified distinct genetic variants associated with RH, including those implicating the role of hyperaldosteronism, highlighting distinct pathways and targets for more effectively treating RH.
Authors
- Anni Kauko (ORCID: https://orcid.org/0000-0003-1131-3865)
- Felix Vaura (ORCID: https://orcid.org/0000-0002-6036-889X)
- Johan Sundström (ORCID: https://orcid.org/0000-0003-2247-8454)
- Sandy Joung (ORCID: https://orcid.org/0000-0002-7582-3218)
- Teemu Niiranen (ORCID: https://orcid.org/0000-0002-7394-7487)
- Joseph E. Ebinger (ORCID: https://orcid.org/0000-0002-0587-1572)
Institutions
- University of Southern California (US)
- Uppsala University (SE)
- University of Turku (FI)
- Turku University of Applied Sciences (FI)
- Turku University Hospital (FI)
- Finnish Institute for Health and Welfare (FI)
- The George Institute for Global Health (AU)
- Department of Medical Sciences (RU)
- Institute for Molecular Medicine Finland (FI)
- Cedars-Sinai Smidt Heart Institute (US)
Publication Details
- Journal
- Hypertension
- Published
- 2025-12-17
- DOI
- https://doi.org/10.1161/hypertensionaha.125.25719
- Citations
- 1
- Primary Topic
- Hormonal Regulation and Hypertension
- Type
- article
- Field-Weighted Citation Impact
- 1.29