Targeting PD-L1 with BMS-202 Enhances Antitumor Cytokine and Cytotoxic T-Lymphocyte Responses in C57BLx/6 Mouse Lung Carcinogenesis
These findings suggest that BMS-202 effectively promotes apoptosis and enhances immune responses in lung cancer, underscoring its potential as a therapeutic agent in treating lung carcinogenesis.
Authors
- Kovan Faidhalla Jalal
- Arash Abdolmaleki (ORCID: https://orcid.org/0000-0002-7454-8728)
- Shang Ziyad Abdulqadir
- Mustafa Fahmi Rajab (ORCID: https://orcid.org/0000-0003-1403-1839)
- Mohammad B. Ghayour
- Christer Janson (ORCID: https://orcid.org/0000-0001-5093-6980)
- Shukur Wasman Smail (ORCID: https://orcid.org/0000-0001-8188-2540)
- Abdullah Abubaker Shareef (ORCID: https://orcid.org/0000-0002-3944-6136)
Institutions
- Uppsala University (SE)
- Cihan University-Erbil (IQ)
- Salahaddin University-Erbil (IQ)
- University of Mohaghegh Ardabili (IR)
- Tishk International University (IQ)
Publication Details
- Journal
- Cancer Management and Research
- Published
- 2025-12-01
- DOI
- https://doi.org/10.2147/cmar.s560799
- Primary Topic
- NF-κB Signaling Pathways
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- Uppsala Universitet
- HjärtLung Uppsala, Länsföreningen