Niacin inhibits vascular calcification via modulating of SIRT1/SIRT6 signaling pathway

Vascular calcification (VC) is a common pathological state that often accompanies calcium-phosphorus metabolism disorder and chronic kidney diseases (CKDs). Vascular smooth muscle cell (VSMC) has been widely acknowledged as one of the main cell types involved in this process. Niacin, a lipid-lowering reagent, has been demonstrated to be beneficial in atherosclerotic disease, but its role in vascular calcification remains unexplored. Restricted cubic spline (RCS) analysis of clinical datasets revealed an inverse correlation between dietary niacin intake and abdominal aortic calcification (AAC). Our data showed that niacin treatment remarkably reduced VSMC osteogenic differentiation. Moreover, niacin treatment alleviated CKD and vitamin D<sub>3</sub>-induced vascular calcification in C57BL/6J mice. Mechanistically, we for the first time demonstrated that niacin inhibited vascular calcification via maintaining both Sirtuin 1 (SIRT1) and Sirtuin 6 (SIRT6) levels. Further, we verified that niacin increased SIRT1 and SIRT6-mediated autophagy flux in VSMC. Our findings reveal that niacin exerts anti-calcification effect via maintaining both SIRT1 and SIRT6, providing novel therapeutic strategies in the treatment of vascular calcification.

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Journal
Cell Death Discovery
Published
2025-12-06
DOI
https://doi.org/10.1038/s41420-025-02882-2
Primary Topic
Sirtuins and Resveratrol in Medicine
Type
article
Field-Weighted Citation Impact
0.00

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article

Niacin inhibits vascular calcification via modulating of SIRT1/SIRT6 signaling pathway

Chow‐Seng Kong, Yi Shi, Yuelin Chao, Li‐Da Wu et al.
Cell Death Discovery
Sirtuins and Resveratrol in Medicine
article

Niacin inhibits vascular calcification via modulating of SIRT1/SIRT6 signaling pathway

Chow‐Seng Kong, Yi Shi, Yuelin Chao, Li‐Da Wu, Jinque Luo, Wenying Zhou, Feng Wang, Yue Gu, Yue Sun, Xiao-min Jiang, Dongchen Wang, Shao-Liang Chen
article en

Abstract

Vascular calcification (VC) is a common pathological state that often accompanies calcium-phosphorus metabolism disorder and chronic kidney diseases (CKDs). Vascular smooth muscle cell (VSMC) has been widely acknowledged as one of the main cell types involved in this process. Niacin, a lipid-lowering reagent, has been demonstrated to be beneficial in atherosclerotic disease, but its role in vascular calcification remains unexplored. Restricted cubic spline (RCS) analysis of clinical datasets revealed an inverse correlation between dietary niacin intake and abdominal aortic calcification (AAC). Our data showed that niacin treatment remarkably reduced VSMC osteogenic differentiation. Moreover, niacin treatment alleviated CKD and vitamin D<sub>3</sub>-induced vascular calcification in C57BL/6J mice. Mechanistically, we for the first time demonstrated that niacin inhibited vascular calcification via maintaining both Sirtuin 1 (SIRT1) and Sirtuin 6 (SIRT6) levels. Further, we verified that niacin increased SIRT1 and SIRT6-mediated autophagy flux in VSMC. Our findings reveal that niacin exerts anti-calcification effect via maintaining both SIRT1 and SIRT6, providing novel therapeutic strategies in the treatment of vascular calcification.

Cell Death DiscoveryVol. 12(1)
Changsha Medical University (CN), Nanjing Medical University (CN)
National Natural Science Foundation of China, Nanjing Medical University
Openalex Percentile: Top 52%
Sirtuins and Resveratrol in Medicine
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