MiR-92a-3p regulates microglial autophagy and inflammation after intracerebral hemorrhage via ATG14

These findings establish miR-92a-3p as a critical regulator of microglial autophagy and inflammation after hemorrhagic stroke and identify it as a prospective therapeutic target for neuroinflammatory modulation.

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Publication Details

Journal
Neurological Research
Published
2025-12-05
DOI
https://doi.org/10.1080/01616412.2025.2594598
Primary Topic
Intracerebral and Subarachnoid Hemorrhage Research
Type
article
Field-Weighted Citation Impact
0.00
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article

MiR-92a-3p regulates microglial autophagy and inflammation after intracerebral hemorrhage via ATG14

Shengyun Hao, Li Tu, Lin Yao, Qiong Mu et al.
Neurological Research
Intracerebral and Subarachnoid Hemorrhage Research
article

MiR-92a-3p regulates microglial autophagy and inflammation after intracerebral hemorrhage via ATG14

Shengyun Hao, Li Tu, Lin Yao, Qiong Mu, Teng Luo, Qian He
article en

Abstract

These findings establish miR-92a-3p as a critical regulator of microglial autophagy and inflammation after hemorrhagic stroke and identify it as a prospective therapeutic target for neuroinflammatory modulation.

Neurological Research
Guiyang Medical University (CN), Tongren Hospital (CN), Affiliated Hospital of Guizhou Medical University (CN), Tongren University (CN)
Openalex Percentile: Top 47%
Intracerebral and Subarachnoid Hemorrhage Research
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MiR-92a-3p regulates microglial autophagy and inflammation after intracerebral hemorrhage via ATG14 — Shengyun Hao, Li Tu, et al. · Neurological Research (2025) | TGRS Research Map | TGRS