Sodium butyrate attenuates early brain injury and neuronal apoptosis after subarachnoid hemorrhage in rats via GPR41/PI3K/Akt pathway

These findings suggest that NaB may mitigate EBI after SAH by inhibiting neuronal apoptosis, with the underlying mechanism potentially involving activation of the GPR41/PI3K/Akt signaling pathway.

Authors

Liang Chen (ORCID: https://orcid.org/0000-0001-6644-477X)
Yufei Ge (ORCID: https://orcid.org/0000-0002-7776-1057)
Kang Liu (ORCID: https://orcid.org/0000-0001-7929-6534)
Meiling Yu

Institutions

Jiangsu Province Hospital (CN)

Publication Details

Journal: Neuroreport
Published: 2025-11-10
DOI: https://doi.org/10.1097/wnr.0000000000002231
Primary Topic: Intracranial Aneurysms: Treatment and Complications
Type: article
Field-Weighted Citation Impact: 0.00

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article

Sodium butyrate attenuates early brain injury and neuronal apoptosis after subarachnoid hemorrhage in rats via GPR41/PI3K/Akt pathway

Liang Chen, Yufei Ge, Kang Liu, Meiling Yu

Nov 10, 2025

Neuroreport

Intracranial Aneurysms: Treatment and Complications

article

Sodium butyrate attenuates early brain injury and neuronal apoptosis after subarachnoid hemorrhage in rats via GPR41/PI3K/Akt pathway

Liang Chen, Yufei Ge, Kang Liu, Meiling Yu

November 10, 2025article en

Abstract

These findings suggest that NaB may mitigate EBI after SAH by inhibiting neuronal apoptosis, with the underlying mechanism potentially involving activation of the GPR41/PI3K/Akt signaling pathway.

NeuroreportVol. 37(1)

Jiangsu Province Hospital (CN)

Openalex Percentile: Top 47%

Intracranial Aneurysms: Treatment and Complications

DOI Source OpenAlex

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Sodium butyrate attenuates early brain injury and neuronal apoptosis after subarachnoid hemorrhage in rats via GPR41/PI3K/Akt pathway — Liang Chen, Yufei Ge, et al. · Neuroreport (2025) | TGRS Research Map | TGRS