GARLH regulates neuroligin preference for excitatory versus inhibitory synapses

Synaptic specificity is governed by precise combinations of cell adhesion proteins that stabilize pre- and postsynaptic sites and appropriate neurotransmitter receptors. The postsynaptic neuroligins NL1/3 and NL2/3/4 localize to excitatory and inhibitory synapses, respectively, and regulate the corresponding neurotransmitter receptors. However, the exact molecular mechanisms that determine synaptic specificity via defined combinations of neuroligins and neurotransmitter receptors remain unclear. We found that all neuroligin isoforms form a tripartite complex with GABAA receptors and GARLH4 protein, with isoform-specific preferences, and that NL1, previously thought to be restricted to excitatory synapses, is also present at inhibitory synapses. In the absence of inhibitory synapse-specific NL2/4, NL1/3 increasingly assembles with GARLH4/GABAA receptors and relocates to inhibitory synapses. Moreover, forced interaction between NL1 and GARLH4 redirects their localization to inhibitory synapses. These findings demonstrate that GARLHs regulate the synaptic specificity of neuroligins, providing the key link between neuroligins and inhibitory GABAA receptors.

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Publication Details

Journal
The Journal of Cell Biology
Published
2025-11-08
DOI
https://doi.org/10.1083/jcb.202507190
Citations
1
Primary Topic
Neuroscience and Neuropharmacology Research
Type
article
Field-Weighted Citation Impact
0.91
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article

GARLH regulates neuroligin preference for excitatory versus inhibitory synapses

Elizabeth J. Salm, Dilja Krueger‐Burg, Tokiwa Yamasaki, Kohtarou Konno et al.
1 citations
The Journal of Cell Biology
Neuroscience and Neuropharmacology Research
0.91
article

GARLH regulates neuroligin preference for excitatory versus inhibitory synapses

Elizabeth J. Salm, Dilja Krueger‐Burg, Tokiwa Yamasaki, Kohtarou Konno, Nashid H. Chaudhury, Susumu Tomita, Megumi Morimoto‐Tomita, Nils Brose, Masahiko Watanabe, Yoav Noam
article en
1 citations

Abstract

Synaptic specificity is governed by precise combinations of cell adhesion proteins that stabilize pre- and postsynaptic sites and appropriate neurotransmitter receptors. The postsynaptic neuroligins NL1/3 and NL2/3/4 localize to excitatory and inhibitory synapses, respectively, and regulate the corresponding neurotransmitter receptors. However, the exact molecular mechanisms that determine synaptic specificity via defined combinations of neuroligins and neurotransmitter receptors remain unclear. We found that all neuroligin isoforms form a tripartite complex with GABAA receptors and GARLH4 protein, with isoform-specific preferences, and that NL1, previously thought to be restricted to excitatory synapses, is also present at inhibitory synapses. In the absence of inhibitory synapse-specific NL2/4, NL1/3 increasingly assembles with GARLH4/GABAA receptors and relocates to inhibitory synapses. Moreover, forced interaction between NL1 and GARLH4 redirects their localization to inhibitory synapses. These findings demonstrate that GARLHs regulate the synaptic specificity of neuroligins, providing the key link between neuroligins and inhibitory GABAA receptors.

The Journal of Cell BiologyVol. 225(2)
Johannes Gutenberg University Mainz (DE), Sapporo University (JP), Hokkaido University (JP), Yale University (US), University Medical Center of the Johannes Gutenberg University Mainz (DE), Max Planck Institute of Experimental Medicine (DE)
Openalex Percentile: Top 20%
Neuroscience and Neuropharmacology Research
0.91
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