<scp>METTL14</scp> Inhibits Human Lung Microvascular Endothelial Cell From Ferroptosis in Septic Lung Injury by Regulating <scp> N <sup>6</sup> </scp> ‐Methyladenosine‐Modified <scp>ACSL4</scp> Level
Our data support that ferroptosis-induced damage is a pathogenic event during septic lung injury and that targeting METTL14 may have therapeutic potential for the treatment of septic lung injury.
Authors
- Rongrong Wu (ORCID: https://orcid.org/0009-0002-7677-4072)
- Gui Xiao (ORCID: https://orcid.org/0000-0001-6001-789X)
- Yongyang Tian
- Fang-yun Gan
- Jinhui Wang (ORCID: https://orcid.org/0000-0001-7024-3313)
- Haiyuan Zhu
- Lilin Wang (ORCID: https://orcid.org/0009-0000-5411-0545)
Institutions
- The Affiliated Yongchuan Hospital of Chongqing Medical University (CN)
- Chongqing Medical University (CN)
Publication Details
- Journal
- Clinical and Experimental Pharmacology and Physiology
- Published
- 2025-11-28
- DOI
- https://doi.org/10.1111/1440-1681.70095
- Primary Topic
- Ferroptosis and cancer prognosis
- Type
- article
- Field-Weighted Citation Impact
- 0.00