Persistently increased CaMKIIδ autophosphorylation mediates pathologic SR Ca loss in a murine model of Doxorubicin-induced cardiomyopathy
Persistently increased CaMKIIδ autophosphorylation, but not oxidation, mediates pathologic SR Ca loss in Doxorubicin-induced cardiomyopathy.
Authors
- Dörthe M. Katschinski (ORCID: https://orcid.org/0000-0003-4630-9081)
- Daniel Tarnowski
- Maria J. Baier (ORCID: https://orcid.org/0000-0003-1722-7976)
- Maithily S. Nanadikar (ORCID: https://orcid.org/0000-0002-5557-8059)
- Anna-Lena Feder
- Can Martin Sag
- Julian Mustroph (ORCID: https://orcid.org/0000-0001-9668-7852)
- Lars Siegfried Maier (ORCID: https://orcid.org/0000-0001-9915-4429)
- Anna-Maria Pfützenreuter
Institutions
- University Hospital Regensburg (DE)
- Universitätsmedizin Göttingen (DE)
- University of Göttingen (DE)
Publication Details
- Journal
- Journal of Molecular and Cellular Cardiology Plus
- Published
- 2025-11-21
- DOI
- https://doi.org/10.1016/j.jmccpl.2025.100828
- Primary Topic
- Chemotherapy-induced cardiotoxicity and mitigation
- Type
- article
- Field-Weighted Citation Impact
- 0.00