Deletion of fibro-adipogenic progenitors-specific follistatin impairs muscle function and accelerates skeletal muscle atrophy in obese mice

Follistatin plays a protective role in mice by maintaining the metabolic health of skeletal muscles; it restores muscle function during HFD challenge, thereby reducing diet-induced obesity-related complications.

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Publication Details

Journal
Molecular Medicine
Published
2025-11-21
DOI
https://doi.org/10.1186/s10020-025-01393-1
Primary Topic
TGF-β signaling in diseases
Type
article
Field-Weighted Citation Impact
0.00
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article

Deletion of fibro-adipogenic progenitors-specific follistatin impairs muscle function and accelerates skeletal muscle atrophy in obese mice

Koichi Ikuta, Shinya Abe, Toshihiko Satake, Yoshiyuki Watanabe et al.
Molecular Medicine
TGF-β signaling in diseases
article

Deletion of fibro-adipogenic progenitors-specific follistatin impairs muscle function and accelerates skeletal muscle atrophy in obese mice

Koichi Ikuta, Shinya Abe, Toshihiko Satake, Yoshiyuki Watanabe, Takumi Era, Sana Khalid, Tomonobu Kado, Ayumi Nishimura, Kaoru Hirabayashi, Hiroyuki Miwa, Martin Matthew Matzuk, Shiho Fujisaka, Masaru Kato, Muhammad Rahil Aslam, Isao Usui, Kazuyuki Tobe, Maki Yokoyama, Yasuhiro Onogi, Naeem Iqbal, Muhammad Bilal, Allah Nawaz, Seiji Yamamoto, Yoshiko Igarashi, Memoona Memoona, Nguyen Quynh Phuong, Le Duc Anh, Kohta Kobayashi
article en

Abstract

Follistatin plays a protective role in mice by maintaining the metabolic health of skeletal muscles; it restores muscle function during HFD challenge, thereby reducing diet-induced obesity-related complications.

Molecular MedicineVol. 31(1)
Joslin Diabetes Center (US), Harvard University (US), Rawalpindi Medical University (PK), Baylor College of Medicine (US), Kyoto University (JP), Phenikaa University (VN), University of Toyama (JP), Dokkyo Medical University (JP), Saitama Medical University (JP), Kumamoto University (JP)
Openalex Percentile: Top 68%
TGF-β signaling in diseases
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