<i>Porphyromonas gingivalis</i> sphingolipids impair neutrophil function and promote bacterial survival
Our data suggest that SL synthesis is an efficient immune evasion mechanism of <i>P. gingivalis</i>, which dampens the inflammatory response of neutrophils to this endogenous pathogen.
Authors
- Fatma Öner (ORCID: https://orcid.org/0000-0001-6193-2029)
- Alpdoğan Kantarcı (ORCID: https://orcid.org/0000-0002-2679-9100)
- Peter A. Zimmerman (ORCID: https://orcid.org/0000-0002-5349-4513)
- Manda Yu (ORCID: https://orcid.org/0000-0001-5932-9597)
- Mary Ellen Davey (ORCID: https://orcid.org/0000-0001-8129-0537)
- Zeliha Güney (ORCID: https://orcid.org/0000-0001-6897-0773)
- Daniel Irimia (ORCID: https://orcid.org/0000-0001-7347-2082)
- Carla Álvarez (ORCID: https://orcid.org/0000-0001-5073-7471)
- Jaime Greatorex
Institutions
- University of Minnesota (US)
- Harvard University (US)
- Ankara University (TR)
- Istanbul Medipol University (TR)
- Massachusetts General Hospital (US)
- Biology of Infection (FR)
- Shriners Hospitals for Children - Boston (US)
- Istinye University (TR)
- Ankara Medipol Üniversitesi
- University at Buffalo, State University of New York (US)
Publication Details
- Journal
- Journal of Oral Microbiology
- Published
- 2025-11-16
- DOI
- https://doi.org/10.1080/20002297.2025.2579103
- Primary Topic
- Sphingolipid Metabolism and Signaling
- Type
- article
- Field-Weighted Citation Impact
- 0.00