MiR-509-3p Targets YAP1 to Regulate Inflammatory Cytokines Against Hyperglycemia-Induced Renal Tubular Cell Damage in Diabetic KidneyDisease

The study preliminarily suggests that miR-509-3p/YAP1 axis may act as a crucial regulator of hyperglycemia-induced tubular inflammation, offering a potential therapeutic target for DKD.

Authors

Institutions

Publication Details

Journal
Combinatorial Chemistry & High Throughput Screening
Published
2025-11-14
DOI
https://doi.org/10.2174/0113862073446094251111052119
Primary Topic
Hippo pathway signaling and YAP/TAZ
Type
article
Field-Weighted Citation Impact
0.00
Controls
|||
ALL TIME
JAN
FEB
MAR
APR
MAY
JUN
JUL
AUG
SEP
OCT
article

MiR-509-3p Targets YAP1 to Regulate Inflammatory Cytokines Against Hyperglycemia-Induced Renal Tubular Cell Damage in Diabetic KidneyDisease

Linlin Li, Xiaoyu Wang, Xiaodong Zhu, Hongdong Li et al.
Combinatorial Chemistry & High Throughput Screening
Hippo pathway signaling and YAP/TAZ
article

MiR-509-3p Targets YAP1 to Regulate Inflammatory Cytokines Against Hyperglycemia-Induced Renal Tubular Cell Damage in Diabetic KidneyDisease

Linlin Li, Xiaoyu Wang, Xiaodong Zhu, Hongdong Li, Shumei Shi, Bowen Tian
article en

Abstract

The study preliminarily suggests that miR-509-3p/YAP1 axis may act as a crucial regulator of hyperglycemia-induced tubular inflammation, offering a potential therapeutic target for DKD.

Combinatorial Chemistry & High Throughput ScreeningVol. 29
People's Liberation Army 401 Hospital (CN)
Openalex Percentile: Top 64%
Hippo pathway signaling and YAP/TAZ
AI Navigator

Ask Laika to Summarize, Analyze, and Connect papers live on the map.

Summarize Papers & Methodologies

Extract key findings, datasets, and comparative methods across publications.

Benchmark Rankings & Visual Analytics

Rank top research institutions, authors, funders, topics, and journals by Field-Weighted Citation Impact (FWCI) and paper volume with instant charts.

Connect Distant Disciplines

Bridge topological clusters on the map to find hidden collaborative intersections.