Botulinum Toxin Type A as a Therapeutic Agent in Epilepsy: Attenuation of Neuronal Ferroptosis and Cognitive Dysfunction
These results suggest that BoNT/A is effective in preventing epileptic neuronal iron death and attenuates cognitive dysfunction through the ferroptosis pathway.
Authors
- Shuang Li (ORCID: https://orcid.org/0009-0009-8865-9267)
- Yajun Lian
- Maolin Du (ORCID: https://orcid.org/0000-0003-3740-0043)
- Zhi Huang (ORCID: https://orcid.org/0000-0001-9980-986X)
- Yake Zheng (ORCID: https://orcid.org/0000-0002-5949-520X)
- Yunqing Ma (ORCID: https://orcid.org/0000-0001-5960-4114)
Institutions
- First Affiliated Hospital of Zhengzhou University (CN)
Publication Details
- Journal
- Brain and Behavior
- Published
- 2025-11-01
- DOI
- https://doi.org/10.1002/brb3.70930
- Primary Topic
- Ferroptosis and cancer prognosis
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Natural Science Foundation of China