Correction to [Microwave‐Responsive Nanoplatform with Robust ROS Generation and Inhibiting Effects of Two‐Component System and Quorum Sensing for the Treatment of MRSA‐Induced Osteomyelitis]

The authors regret the misuse of immunofluorescence staining pictures for TNF- and IL-10 in rat wounds at 4 hours in Figure 6e (Mn 0.1 PCC+MW) and Figure 6g (Mn 0.1 PCC).The modified correct version is shown below.In this study, both bacterial infection and ROS promote the shift of macrophages to the pro-inflammatory M1 type.As shown in Figure 6d, the Mn 0.1 PCC group has a weaker antibacterial capacity than the Mn 0.1 PCC+MW group, and a large number of bacteria are still present at the site of bacterial infection.At the same time, a large amount of ROS is produced by Mn 0.1 PCC.Therefore, the presence of bacteria and the production of ROS in the Mn 0.1 PCC group promote a pro-inflammatory M1-type shift of macrophages at the wound site.To systematically quantify this phenotypic shift, the fluorescence intensities of TNF- and IL-10 in Figure 6e,g are calculated, and the results are shown in Figure 6f,h.The expression level of the pro-inflammatory factor TNF- is increased in the Mn 0.1 PCC group, while the expression level of the anti-inflammatory factor IL-10 is decreased in the Mn 0.1 PCC group.Figure 6.Immunofluorescence staining of macrophages of e) TNF- and g) IL-10 after 4 h (scale bar = 100 m); f,h) Mean fluorescence intensity of (f) TNF- and (h) IL-10.(Control) represents the rat without any treatment; (Control + MW) represents the rat treated with MW irradiation; (Mn 0.1 PCC) represents the rat after treatment with Mn 0.1 PCC; (Mn 0.1 PCC + MW) represents the rat after treatment with Mn 0.1 PCC and MW irradiation.We apologize for this error.