Cms1 Ribosomal Small Subunit Homolog Promotes HCC Proliferation and Migration by Modulating the TNF/NF-κB Signaling Pathway
The study suggested that CMSS1 could be a critical modulator of HCC tumorigenesis and metastasis through the TNF/NF-κB signaling pathway, thus being considered as a potential therapeutic target for HCC. Targeting CMSS1 may offer a novel strategy to inhibit NF-κB-driven inflammatory signaling and suppress tumor progression in HCC.
Authors
- Peng Zhang (ORCID: https://orcid.org/0000-0002-7739-6109)
- Yongjie Yu
- Sha Hu
- Haitao Wang (ORCID: https://orcid.org/0000-0003-0338-4364)
- Yu Zhang (ORCID: https://orcid.org/0000-0001-8346-2179)
- Tuo Zhang (ORCID: https://orcid.org/0000-0002-4056-3815)
- Zeming Cai
- Yongping Huang
- Jing Dai (ORCID: https://orcid.org/0009-0000-6866-0986)
- Qin Fang (ORCID: https://orcid.org/0009-0005-7530-9787)
Institutions
- Wuhan University (CN)
- Renmin Hospital of Wuhan University (CN)
Publication Details
- Journal
- Journal of Hepatocellular Carcinoma
- Published
- 2025-10-01
- DOI
- https://doi.org/10.2147/jhc.s549003
- Primary Topic
- Signaling Pathways in Disease
- Type
- article
- Field-Weighted Citation Impact
- 0.00