TPEN attenuates neuronal ferroptosis in ischemic brain injury via GPR39-mediated PI3K/AKT signaling activation

TPEN attenuates neuronal ferroptosis an brain injury by activating the GPR39/PI3K/AKT signaling axis. This identifies GPR39 as a novel therapeutic target for brain injury treatment.

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Publication Details

Journal
Neurological Research
Published
2025-10-21
DOI
https://doi.org/10.1080/01616412.2025.2578343
Primary Topic
Ferroptosis and cancer prognosis
Type
article
Field-Weighted Citation Impact
0.00
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article

TPEN attenuates neuronal ferroptosis in ischemic brain injury via GPR39-mediated PI3K/AKT signaling activation

Zhenhong Li, Fei Li
Neurological Research
Ferroptosis and cancer prognosis
article

TPEN attenuates neuronal ferroptosis in ischemic brain injury via GPR39-mediated PI3K/AKT signaling activation

Zhenhong Li, Fei Li
article en

Abstract

TPEN attenuates neuronal ferroptosis an brain injury by activating the GPR39/PI3K/AKT signaling axis. This identifies GPR39 as a novel therapeutic target for brain injury treatment.

Neurological Research
Ganzhou People's Hospital (CN)
Openalex Percentile: Top 57%
Ferroptosis and cancer prognosis
DOISourceOpenAlex
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TPEN attenuates neuronal ferroptosis in ischemic brain injury via GPR39-mediated PI3K/AKT signaling activation — Zhenhong Li, Fei Li · Neurological Research (2025) | TGRS Research Map | TGRS