BMAL1 alleviates sepsis-induced acute kidney injury by inhibiting apoptosis, ferroptosis and inflammation
BMAL1 overexpression mediated by USP10-induced deubiquitination or HOXA5-induced transcription can attenuate sepsis-induced acute kidney injury, recommending a novel insight for the prevention of sepsis-induced AKI.
Authors
- Zhi Chen (ORCID: https://orcid.org/0000-0003-2095-1324)
- Jinhui Shi (ORCID: https://orcid.org/0000-0001-8894-3967)
- Litong Jin (ORCID: https://orcid.org/0000-0001-6181-6650)
- Gang Chen (ORCID: https://orcid.org/0000-0002-8320-6594)
Institutions
- Taizhou Central Hospital (CN)
Publication Details
- Journal
- Hereditas
- Published
- 2025-10-14
- DOI
- https://doi.org/10.1186/s41065-025-00583-5
- Primary Topic
- Heme Oxygenase-1 and Carbon Monoxide
- Type
- article
- Field-Weighted Citation Impact
- 0.00