Inhibition of S100A9 Improves Aortic Dissection in Association With Mitochondrial Function Enhancement
S100A9 is a potential contributor to AD pathogenesis. Inhibition of S100A9 might be a promising therapeutic target for AD.
Authors
- Yiying Zhang (ORCID: https://orcid.org/0000-0002-4280-4811)
- Kai Guo (ORCID: https://orcid.org/0000-0003-0384-4754)
- Xiao-Wen Wang (ORCID: https://orcid.org/0000-0002-6106-8186)
- Man Pun Wan (ORCID: https://orcid.org/0000-0002-5326-6705)
- Yongzheng Guo (ORCID: https://orcid.org/0000-0003-4981-8957)
- Linman Li (ORCID: https://orcid.org/0000-0002-2757-7083)
- Zhao Zhang (ORCID: https://orcid.org/0000-0001-5320-2210)
- Keyu Zhang (ORCID: https://orcid.org/0000-0001-5296-9735)
- Yu Zhao (ORCID: https://orcid.org/0000-0003-4035-0825)
Institutions
- Jiamusi University (CN)
- The Affiliated Yongchuan Hospital of Chongqing Medical University (CN)
- Chongqing Medical University (CN)
Publication Details
- Journal
- Frontiers in Bioscience-Landmark
- Published
- 2025-09-29
- DOI
- https://doi.org/10.31083/fbl44666
- Primary Topic
- S100 Proteins and Annexins
- Type
- article
- Field-Weighted Citation Impact
- 0.00