An Elusive Diagnosis of Autosomal Dominant Alport Syndrome: Genomic Sequencing Is a Game Changer

We report the case of a 55-year-old woman with chronic asymptomatic hematuria and a longstanding diagnosis of thin basement membrane disease, presenting with worsening hypertension and a significant degree of proteinuria progressing to chronic kidney disease. Genetic sequencing identified a heterozygous pathogenic variant in the COL4A4 gene (c.1321_1369+3del), a 52-base pair deletion that disrupts normal ribonucleic acid (RNA) splicing in the exon 20/intron 20 junction. In silico analysis predicted the complete loss of the canonical splice site, leading to improper splicing and the dysfunction of the α4 chain of type-IV collagen. Genetic testing in the family confirmed the presence of the same variant in two additional generations. To our knowledge, this is the first reported family in which the c.1321_1369+3del mutation is the sole cause of autosomal dominant Alport syndrome.

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Publication Details

Journal
Cureus
Published
2025-10-07
DOI
https://doi.org/10.7759/cureus.94077
Primary Topic
Cell Adhesion Molecules Research
Type
article
Field-Weighted Citation Impact
0.00
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article

An Elusive Diagnosis of Autosomal Dominant Alport Syndrome: Genomic Sequencing Is a Game Changer

Nikolaos Sabanis, Achilleas Betsikos, Eleni Paschou, Virginia Geladari et al.
Cureus
Cell Adhesion Molecules Research
article

An Elusive Diagnosis of Autosomal Dominant Alport Syndrome: Genomic Sequencing Is a Game Changer

Nikolaos Sabanis, Achilleas Betsikos, Eleni Paschou, Virginia Geladari, Evanthia Gazouni
article en

Abstract

We report the case of a 55-year-old woman with chronic asymptomatic hematuria and a longstanding diagnosis of thin basement membrane disease, presenting with worsening hypertension and a significant degree of proteinuria progressing to chronic kidney disease. Genetic sequencing identified a heterozygous pathogenic variant in the COL4A4 gene (c.1321_1369+3del), a 52-base pair deletion that disrupts normal ribonucleic acid (RNA) splicing in the exon 20/intron 20 junction. In silico analysis predicted the complete loss of the canonical splice site, leading to improper splicing and the dysfunction of the α4 chain of type-IV collagen. Genetic testing in the family confirmed the presence of the same variant in two additional generations. To our knowledge, this is the first reported family in which the c.1321_1369+3del mutation is the sole cause of autosomal dominant Alport syndrome.

CureusVol. 17(10)
University Hospital of Larissa (GR), e-Trikala (Greece) (GR)
Openalex Percentile: Top 64%
Cell Adhesion Molecules Research
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