Comparable expression of stem markers and damage-responsive proteins in untreated and chemoradiation-treated rectal cancer.

PURPOSE: Cancer stem cells (CSCs) are suggested to contribute to therapy resistance in rectal cancer. However, histopathological analysis of CSCs is still lacking in rectal cancer. METHODS: hybridization and immunostaining. Niche factors were also compared in human rectal cancer specimens and the embryonic intestine. RESULTS: The mean ratios were 15% and 14% for LGR5, 30% and 33% for BMI1, 2.7% and 7.6% for YAP/TAZ, and 38% and 32% for nuclear β-catenin in untreated and CRT rectal cancer, respectively, showing no significant differences for these stem markers following CRT. The stromal expression ratios of YAP/TAZ were 9.7% and 23% in untreated and CRT rectal cancer, which indicated significant upregulation and confirmation of the damage response in the stroma of CRT tumors. In addition, cancer cells co-expressing LGR5 and CDKN1B are also comparable in untreated and CRT rectal cancer. For niche factors, we found that WNT2B and GREM1 were uniformly expressed in rectal cancer with a pattern similar to that of the early embryonic intestine. CONCLUSION: The expression of LGR5, BMI1, CDKN1B, and YAP/TAZ was comparable in untreated and CRT rectal cancer. The uniform expression of mesenchymal niche factors might prevent the zonation of the stem cell niche in rectal cancer.

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Journal
PubMed
Published
2026-04-01
DOI
https://doi.org/10.14670/hh-18-999
Primary Topic
Cancer Cells and Metastasis
Type
article
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article

Comparable expression of stem markers and damage-responsive proteins in untreated and chemoradiation-treated rectal cancer.

Tomoko Kamiakito, Hirotoshi Kawata, Takeo Nakaya, Sachi Sekine et al.
PubMed
Cancer Cells and Metastasis
article

Comparable expression of stem markers and damage-responsive proteins in untreated and chemoradiation-treated rectal cancer.

Tomoko Kamiakito, Hirotoshi Kawata, Takeo Nakaya, Sachi Sekine, Yasuyuki Miyakura, Koichi Suzuki, Akira Tanaka, Kentaro Tsuji, Toshiki Rikiyama
article en

Abstract

PURPOSE: Cancer stem cells (CSCs) are suggested to contribute to therapy resistance in rectal cancer. However, histopathological analysis of CSCs is still lacking in rectal cancer. METHODS: hybridization and immunostaining. Niche factors were also compared in human rectal cancer specimens and the embryonic intestine. RESULTS: The mean ratios were 15% and 14% for LGR5, 30% and 33% for BMI1, 2.7% and 7.6% for YAP/TAZ, and 38% and 32% for nuclear β-catenin in untreated and CRT rectal cancer, respectively, showing no significant differences for these stem markers following CRT. The stromal expression ratios of YAP/TAZ were 9.7% and 23% in untreated and CRT rectal cancer, which indicated significant upregulation and confirmation of the damage response in the stroma of CRT tumors. In addition, cancer cells co-expressing LGR5 and CDKN1B are also comparable in untreated and CRT rectal cancer. For niche factors, we found that WNT2B and GREM1 were uniformly expressed in rectal cancer with a pattern similar to that of the early embryonic intestine. CONCLUSION: The expression of LGR5, BMI1, CDKN1B, and YAP/TAZ was comparable in untreated and CRT rectal cancer. The uniform expression of mesenchymal niche factors might prevent the zonation of the stem cell niche in rectal cancer.

PubMedVol. 41(4)
Jichi Medical University (JP), Jichi Medical University Saitama Medical Center (JP)
Openalex Percentile: Top 100%
Cancer Cells and Metastasis
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