Ailanthone induces apoptosis in U-2OS cells through the endoplasmic reticulum stress
AIL inhibited the proliferation of human OS cells and induced apoptosis through the ERS pathway. It represented a potential therapeutic agent for OS treatment.
Authors
- Lei Liu (ORCID: https://orcid.org/0000-0002-3710-7042)
- Taiding Wu
- Jingxian Li (ORCID: https://orcid.org/0000-0003-1087-5428)
- Rui-Ting Li (ORCID: https://orcid.org/0000-0002-4720-6255)
- Yue Zhang (ORCID: https://orcid.org/0000-0001-6349-6849)
- Jun Wang (ORCID: https://orcid.org/0000-0002-0578-046X)
- Chang Li (ORCID: https://orcid.org/0000-0003-0195-1003)
- Jingyi Zhang (ORCID: https://orcid.org/0000-0002-2844-3431)
- Na Luo (ORCID: https://orcid.org/0000-0002-7919-2142)
- Min Tian (ORCID: https://orcid.org/0000-0002-6157-9074)
Institutions
- Minzu University of China (CN)
- Jianghan University (CN)
- Minda Hospital (CN)
- Wuhan Union Hospital (CN)
- Union Hospital (CN)
Publication Details
- Journal
- Frontiers in Immunology
- Published
- 2025-10-02
- DOI
- https://doi.org/10.3389/fimmu.2025.1633643
- Primary Topic
- Endoplasmic Reticulum Stress and Disease
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Natural Science Foundation of China