Oxidative stress-mediated apoptosis via the SLC23A2-ascorbic acid interaction contributes to cleft lip development

<i>SLC23A2</i> was significantly associated with NSCLO. The <i>SLC23A2</i>/exogenous ROS/AA interaction is involved in lip and craniofacial development by influencing the levels of ROS and apoptosis.

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Publication Details

Journal
Frontiers in Pediatrics
Published
2025-10-02
DOI
https://doi.org/10.3389/fped.2025.1632778
Primary Topic
Cleft Lip and Palate Research
Type
article
Field-Weighted Citation Impact
0.00
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article

Oxidative stress-mediated apoptosis via the SLC23A2-ascorbic acid interaction contributes to cleft lip development

Bin‐Cheng Yin, Yi Xu, Yong Lu
Frontiers in Pediatrics
Cleft Lip and Palate Research
article

Oxidative stress-mediated apoptosis via the SLC23A2-ascorbic acid interaction contributes to cleft lip development

Bin‐Cheng Yin, Yi Xu, Yong Lu
article en

Abstract

<i>SLC23A2</i> was significantly associated with NSCLO. The <i>SLC23A2</i>/exogenous ROS/AA interaction is involved in lip and craniofacial development by influencing the levels of ROS and apoptosis.

Frontiers in PediatricsVol. 13
Sichuan University (CN), Stomatology Hospital (CN), Nanjing Medical University (CN)
Openalex Percentile: Top 71%
Cleft Lip and Palate Research
DOIOpen Access PDFOpenAlex
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