Thymidine exerts anti-doxorubicin-induced cardiomyopathy effect through the regulation of the PPAR signaling pathways and ferroptosis pathways

Thy may exert its anti-DOX-induced cardiomyopathy effect through the regulation of the PPAR signaling and ferroptosis pathways.

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Publication Details

Journal
Frontiers in Pharmacology
Published
2025-09-29
DOI
https://doi.org/10.3389/fphar.2025.1524167
Primary Topic
Congenital heart defects research
Type
article
Field-Weighted Citation Impact
0.00
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article

Thymidine exerts anti-doxorubicin-induced cardiomyopathy effect through the regulation of the PPAR signaling pathways and ferroptosis pathways

Chen Sun, Peihai Li, Yun Liang Zhang, Bin Li et al.
Frontiers in Pharmacology
Congenital heart defects research
article

Thymidine exerts anti-doxorubicin-induced cardiomyopathy effect through the regulation of the PPAR signaling pathways and ferroptosis pathways

Chen Sun, Peihai Li, Yun Liang Zhang, Bin Li, Qifei Wang, Juan Zhou, Qing Xia
article en

Abstract

Thy may exert its anti-DOX-induced cardiomyopathy effect through the regulation of the PPAR signaling and ferroptosis pathways.

Frontiers in PharmacologyVol. 16
Qilu University of Technology (CN), Shandong Iron and Steel Group (China) (CN), Shandong Academy of Sciences (CN)
Openalex Percentile: Top 76%
Congenital heart defects research
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Thymidine exerts anti-doxorubicin-induced cardiomyopathy effect through the regulation of the PPAR signaling pathways and ferroptosis pathways — Chen Sun, Peihai Li, et al. · Frontiers in Pharmacology (2025) | TGRS Research Map | TGRS