INHBA knockdown inhibits renal fibrosis in mice following ischemia–reperfusion injury by suppressing activation of the TGF-β/Smad signaling pathway

Knockdown of INHBA attenuates renal fibrosis after I/R injury in mice by suppressing activation of the TGF-β/Smad signaling pathway. This pathway may represent a potential therapeutic target for renal I/R injury.

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Publication Details

Journal
BMC Nephrology
Published
2025-09-26
DOI
https://doi.org/10.1186/s12882-025-04443-2
Citations
3
Primary Topic
Acute Kidney Injury Research
Type
article
Field-Weighted Citation Impact
4.29
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article

INHBA knockdown inhibits renal fibrosis in mice following ischemia–reperfusion injury by suppressing activation of the TGF-β/Smad signaling pathway

Qian Sun, Qiao Tang, Yifei Wang
3 citations
BMC Nephrology
Acute Kidney Injury Research
4.29
article

INHBA knockdown inhibits renal fibrosis in mice following ischemia–reperfusion injury by suppressing activation of the TGF-β/Smad signaling pathway

Qian Sun, Qiao Tang, Yifei Wang
article en
3 citations

Abstract

Knockdown of INHBA attenuates renal fibrosis after I/R injury in mice by suppressing activation of the TGF-β/Smad signaling pathway. This pathway may represent a potential therapeutic target for renal I/R injury.

BMC NephrologyVol. 26(1)
Wuhan University (CN), Wuhan College (CN), Wuchang University of Technology (CN), Renmin Hospital of Wuhan University (CN)
Openalex Percentile: Top 5%
Acute Kidney Injury Research
4.29
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INHBA knockdown inhibits renal fibrosis in mice following ischemia–reperfusion injury by suppressing activation of the TGF-β/Smad signaling pathway — Qian Sun, Qiao Tang, et al. · BMC Nephrology (2025) | TGRS Research Map | TGRS