The Role of <scp>P62</scp>/Nrf2/Keap1 Signaling Pathway in Lead‐Induced Neurological Dysfunction

The P62/Nrf2/Keap1 pathway mediates Pb-induced neuronal dysfunction and highlights its potential as a therapeutic target for mitigating the neurodegenerative effects associated with Pb exposure.

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Publication Details

Journal
CNS Neuroscience & Therapeutics
Published
2025-09-01
DOI
https://doi.org/10.1111/cns.70566
Citations
3
Primary Topic
Heavy Metal Exposure and Toxicity
Type
article
Field-Weighted Citation Impact
3.60

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article

The Role of P62/Nrf2/Keap1 Signaling Pathway in Lead‐Induced Neurological Dysfunction

Ruokun Wei, Dongjie Peng, Zhenning Li, Shaojun Li et al.
3 citations
CNS Neuroscience & Therapeutics
Heavy Metal Exposure and Toxicity
3.60
article

The Role of P62/Nrf2/Keap1 Signaling Pathway in Lead‐Induced Neurological Dysfunction

Ruokun Wei, Dongjie Peng, Zhenning Li, Shaojun Li, Huishuai Li, Peiqi Wei
article en
3 citations

Abstract

The P62/Nrf2/Keap1 pathway mediates Pb-induced neuronal dysfunction and highlights its potential as a therapeutic target for mitigating the neurodegenerative effects associated with Pb exposure.

CNS Neuroscience & TherapeuticsVol. 31(9)
Guangxi Medical University (CN)
National Natural Science Foundation of China
Openalex Percentile: Top 7%
Heavy Metal Exposure and Toxicity
3.60
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The Role of <scp>P62</scp>/Nrf2/Keap1 Signaling Pathway in Lead‐Induced Neurological Dysfunction — Ruokun Wei, Dongjie Peng, et al. · CNS Neuroscience & Therapeutics (2025) | TGRS Research Map | TGRS