Pathogenic Glycosyltransferase Genes and Potential Therapeutic Drugs in Pressure Overload-Induced Heart Failure

Dysregulation of GTG expression may affect TAC-induced HF through metabolic pathways, and GXYLT2 may be a new potential therapeutic target for HF.

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Publication Details

Journal
ESC Heart Failure
Published
2025-09-03
DOI
https://doi.org/10.1002/ehf2.15409
Primary Topic
Galectins and Cancer Biology
Type
article
Field-Weighted Citation Impact
0.00

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article

Pathogenic Glycosyltransferase Genes and Potential Therapeutic Drugs in Pressure Overload-Induced Heart Failure

Chenze Li, Xiaorong Hu, Zhibing Lu, Xuemei Gao et al.
ESC Heart Failure
Galectins and Cancer Biology
article

Pathogenic Glycosyltransferase Genes and Potential Therapeutic Drugs in Pressure Overload-Induced Heart Failure

Chenze Li, Xiaorong Hu, Zhibing Lu, Xuemei Gao, Yi Lu, Jiahe Wu
article en

Abstract

Dysregulation of GTG expression may affect TAC-induced HF through metabolic pathways, and GXYLT2 may be a new potential therapeutic target for HF.

ESC Heart FailureVol. 12(6)
Wuhan University (CN), Zhongnan Hospital of Wuhan University (CN)
National Natural Science Foundation of China, Fundamental Research Funds for the Central Universities
Openalex Percentile: Top 75%
Galectins and Cancer Biology
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