Mitochondria-target ubiquinone attenuates bleomycin-induced pulmonary fibrosis
These findings not only advance our understanding of the interplay between oxidative stress and pro-fibrotic signaling in bleomycin-induced pulmonary fibrosis but also provide experimental data supporting the use of mitochondria-targeted antioxidant in the treatment of this condition.
Authors
- Xiufang Weng (ORCID: https://orcid.org/0000-0003-2625-2137)
- Zhenghui Huang (ORCID: https://orcid.org/0009-0004-2883-5197)
- Mi Wu (ORCID: https://orcid.org/0000-0003-3774-9624)
- Rui Wang (ORCID: https://orcid.org/0000-0001-7015-0272)
- Ying Jiang (ORCID: https://orcid.org/0009-0002-0642-4106)
- Limin Chen (ORCID: https://orcid.org/0000-0002-7370-6499)
- Lan Lin
- Juan Zhao (ORCID: https://orcid.org/0000-0001-8796-8521)
- Ting Zhou (ORCID: https://orcid.org/0000-0003-3199-4354)
- Zhengfeng Xiong (ORCID: https://orcid.org/0009-0001-4895-3201)
Institutions
- Fujian Medical University (CN)
- Ministry of Education of the People's Republic of China (CN)
- Chinese Academy of Medical Sciences & Peking Union Medical College (CN)
- Union Hospital (CN)
- Huazhong University of Science and Technology (CN)
Publication Details
- Journal
- Frontiers in Pharmacology
- Published
- 2025-09-04
- DOI
- https://doi.org/10.3389/fphar.2025.1661644
- Citations
- 2
- Primary Topic
- Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
- Type
- article
- Field-Weighted Citation Impact
- 3.04
Funders
- National Health Commission of the People's Republic of China