TRPV6-mediated Store-operated Ca2+ Entry Participates in Pancreatic Acinar Cell Injury During Acute Pancreatitis
TRPV6 is a key player in Ca<sup>2+</sup> overload in PACs and contributes to AP at least partly through mediating SOCE.
Authors
- Dan Du (ORCID: https://orcid.org/0000-0001-9745-8197)
- Zhenlu Li (ORCID: https://orcid.org/0000-0003-2101-8237)
- Yuncheng Luo
- Chenxia Han (ORCID: https://orcid.org/0000-0003-1727-7236)
- Wei Huang (ORCID: https://orcid.org/0000-0001-7004-6408)
- Shiyu Liu (ORCID: https://orcid.org/0000-0001-7972-4527)
- Li Li (ORCID: https://orcid.org/0000-0002-0588-0233)
- Qing Xia (ORCID: https://orcid.org/0000-0003-4373-2722)
- Tingting Liu (ORCID: https://orcid.org/0000-0003-4183-0564)
- Ruotian Jiang (ORCID: https://orcid.org/0000-0001-5322-6802)
- Jiawang Li (ORCID: https://orcid.org/0000-0001-6120-2915)
- Juan Lin (ORCID: https://orcid.org/0000-0001-5143-8158)
- Lu Li (ORCID: https://orcid.org/0009-0001-6167-3765)
- Ping Liao
Institutions
- University of Liverpool (GB)
- Sichuan University (CN)
- West China Hospital of Sichuan University (CN)
Publication Details
- Journal
- Cellular and Molecular Gastroenterology and Hepatology
- Published
- 2025-01-01
- DOI
- https://doi.org/10.1016/j.jcmgh.2025.101620
- Citations
- 1
- Primary Topic
- Pancreatitis Pathology and Treatment
- Type
- article
- Field-Weighted Citation Impact
- 2.01