Med13 and Med13L: Critical redundant players in basal cardiac function and gene expression

Med13 and Med13L function partially redundantly within the heart to maintain basal cardiac function and transcription, as well as redundancies within cardiac phenotypes related to mediator complex disruptions.

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Publication Details

Journal
Journal of Molecular and Cellular Cardiology Plus
Published
2025-08-31
DOI
https://doi.org/10.1016/j.jmccpl.2025.100481
Primary Topic
Cardiac Fibrosis and Remodeling
Type
article
Field-Weighted Citation Impact
0.00
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article

Med13 and Med13L: Critical redundant players in basal cardiac function and gene expression

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Med13 and Med13L: Critical redundant players in basal cardiac function and gene expression

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article en

Abstract

Med13 and Med13L function partially redundantly within the heart to maintain basal cardiac function and transcription, as well as redundancies within cardiac phenotypes related to mediator complex disruptions.

Journal of Molecular and Cellular Cardiology PlusVol. 13
University of Iowa (US), Fraternal Order of Eagles (US), The Ohio State University Wexner Medical Center (US)
Openalex Percentile: Top 64%
Cardiac Fibrosis and Remodeling
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